The higher the level of glucose in our blood, the higher the amount of insulin released. is the Chief Medical Officer of West-Martin Longevity. When it comes to sugar and weight issues, it really is that simple. Over time, and given enough fructose, fat droplets start to accumulate in the liver cells, which ultimately results in non-alcoholic liver disease – so called because the effect is much the same as that which alcohol has on the liver. Perhaps the least publicised health complaint on our list is fatty liver disease. Let’s start then by clarifying exactly what is “too much” sugar. Further research by America’s National Center for Biotechnology Information found that those who ate a diet with a high glycemic load (GL) increased their chances of developing prostate cancer by almost 30 per cent, rectal cancer by 44 per cent, and pancreatic cancer by 41 per cent. Those who get 17-21% of their calories from sugars are at a 38% higher risk of dying from cardiovascular disease than those who consume just 8% of their calories from sugar. He is a Consultant to Cenegenics, Inc. However, high fructose levels can block the transport of leptin from the blood to the brain – making it incorrectly believe that the body needs to eat more and burn less in order to replenish our fat stores. . Researchers from the Stanford University School of Medicine, the University of California-Berkeley and the University of California-San Francisco examined data on sugar availability and diabetes rates from 175 countries over the past decade and found evidence to suggest a direct link between the two. The reality is that the link is very easy to explain: If you consume a diet consistently high in sugar (150 pounds per year) and grains (200 pounds per year) and processed foods (full of sugars and trans fats), your blood glucose levels will be chronically elevated. However, high fructose levels can block the transport of leptin from the blood to the brain – making it incorrectly believe that the body needs to eat more and burn less in order to replenish our fat stores. According to statistics from 2014, one in five of us here in the Emirates is diabetic – and the rest of the world doesn’t fare much better either. In the United States, for example, that figure tops 120g per day. A medium-sized banana contains upwards of 20 grams. It is safe to assume that the figures for several Gulf countries, including the UAE, are likely to be quite high too. Over time, this can lead to a greater accumulation of fat, which may turn into fatty liver disease, a … One such study published in the Journal of Clinical Investigation points to sugar not only being a fuel source for existing cancers, but also cites it as a primary factor in the initiation of cancerous characteristics in previously healthy cells. And the reality is that the link is very easy to explain: If you consume a diet consistently high in sugar (150 pounds per year) and grains (200 pounds per year) and processed foods (full of sugars and trans fats), your blood glucose levels will be chronically elevated. Health for many years and remains committed to delivering Proactive Health to physicians and clients around the world. Cardiovascular disease is a leading cause of death here in the UAE (occurring 15-20 years earlier than the global average), and once again our high sugar intake is the biggest contributor to … Clearly then, this is about more than a couple of extra cavities and a few extra pounds. Recent studies show that sugar is responsible for 50% of all healthcare costs! The paper went on to call Alzheimer’s, ‘type 3 diabetes’ – a term now very commonly used among medical professionals. Once again, this particular problem is caused by your body's least favourite sugar – fructose. DiabetesAccording to statistics from 2014, one in five of us here in the Emirates is diabetic – and the rest of the world doesn’t fare much better either. A study published in the Journal of Clinical Investigation points to sugar not only being a fuel source for existing cancers, but also cites it as a primary factor in the initiation of cancerous characteristics in previously healthy cells.

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